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PTD-DBM

Aliases: Protein Transduction Domain-Dvl Binding Motif

Last verified: 2026-09-24

Research chemicalPreclinical evidencePeptidesLow interest

The short version

PTD-DBM is known in the gray hair-growth story for one detail: in mouse models it worked topically in combination with valproic acid, and valproate alone has proven follicle effects. What share of the effect belongs to the peptide — nobody knows. In addition, the Wnt pathway is a double-edged sword: the same signal that drives follicular regeneration is the one wrongly activated in colorectal cancers. The cosmetics industry long ago chose safer Wnt modulators; gray PTD-DBM remains a laboratory reagent with an interesting bibliography and an improper application.

Identity & type

Molecular type
hybrid peptide
Origin
Designed: PTD (transduction domain) + DBM motif (Dvl binding); synthetic.

Mechanism of action

A hybrid peptide: a protein-transduction domain (cell entry) fused to a Dvl-binding motif that blocks the CXXC5–Dishevelled interaction; the result is released Wnt/beta-catenin signaling — the central pathway for the follicular cycle and osteogenesis.

not confirmed in humans

Dosing & routes

Official / clinical context

No official context exists.

Regulator-approved labeling and published study designs — never a recommendation.

Community-reported practice

Occasionally in experimental 'hair stack' protocols (topical, with microneedling), without published measurements; reports are inseparable from other routine components.

Unverified self-reports. Not medical advice. Not endorsement.

Protocol — official vs community

Official / label

No approved protocol exists — preclinical agent. PTD-DBM has only mouse hair- and bone-regeneration data (topical/injectable µg–mg doses); no human regimen exists.

Duration:

The mechanism itself is the caution: chronic Wnt/beta-catenin activation is tumor-associated, so even a perfected formulation would carry a fundamental question.

Community (anecdotal)

Structured protocol data for this entry is coming.

Human evidence

None.

Preclinical evidence

One tidy line of mouse studies from one group; reproduced at least once independently for the follicular model, but without larger scale.

Known risks

  • Theoretical: non-selective Wnt pathway activationtheoretical
  • Complete uncertainty of topical-form bioavailability in humanstheoretical

Teal: characterized in clinical/labeling contexts. Amber: theoretical or reported outside controlled settings.

Unknowns & evidence gaps

  • Everything in humans — from skin penetration to effect

Frequently asked questions

References

  1. Lee SH et al. — PTD-DBM and valproic acid in hair regrowth (mouse models, 2015–2017)